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ProteomicsATM promotes reversed fork processing during DNA interstrand cross-link repair
During replication-coupled DNA interstrand cross-link (ICL) repair, fork reversal is thought to enable the Fanconi anemia (FA) pathway to resolve the ICL through nucleolytic incisions. Subsequent fork restoration then allows nascent DNA strand extension past the lesion. Although these fork remodelin...
ORGANISM(S): Xenopus laevis (African clawed frog) 
2026-08-25 | PXD071242 | Pride
Fork reversal Interstrand cross-link Exo1 Icl repair Pp2a Fanconi anemia pathway Dna2 Atm Fork restoration Orbitrap astral mass spectrometer Wrn 
UnknownOxygen independent DNA interstrand cross-link formation by a nucleotide radical.
Not available
S-EPMC1752237 | biostudies-literature
UnknownEffect of cross-link structure on DNA interstrand cross-link repair synthesis.
Not available
S-EPMC2746369 | biostudies-literature
UnknownDouble-strand break formation during nucleotide excision repair of a DNA interstrand cross-link.
Not available
S-EPMC2725361 | biostudies-literature
UnknownCross-link structure affects replication-independent DNA interstrand cross-link repair in mammalian cells.
Not available
S-EPMC2864352 | biostudies-literature
UnknownThe HMCES DNA-protein cross-link functions as an intermediate in DNA interstrand cross-link repair.
Not available
S-EPMC9949344 | biostudies-literature
UnknownMechanism and regulation of incisions during DNA interstrand cross-link repair.
Not available
S-EPMC4076290 | biostudies-literature
UnknownPhoto-Reactivity of Binaphthalene Triphenylphosphonium Salts: DNA Interstrand Cross-Link Formation and Substituent Effects.
Not available
S-EPMC12398330 | biostudies-literature
UnknownInterstrand cross-link formation in duplex and triplex DNA by modified pyrimidines.
Not available
S-EPMC2556550 | biostudies-literature
GenomicsBRCA1 functions independently of homologous recombination in DNA interstrand crosslink repair
Brca1 is required for DNA repair by homologous recombination (HR) and normal embryonic development. Here we report that deletion of the DNA damage responsefactor 53BP1 overcomes embryonic lethality in Brca1-nullizygous mice, and rescues HR deficiency, as measured by hypersensitivity to PARP (polyADP...
ORGANISM(S): Mus musculus 
2012-09-30 | E-GEOD-35698 | biostudies-arrayexpress
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