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UnknownStraightforward Access to Polyfunctionalized δ-Lactams via Domino Aza-Michael/Thia-Michael/Aldol Sequence.
Not available
S-EPMC12114351 | biostudies-literature
UnknownStaged Screening Identifies People with Biomarkers Related to Neuronal Alpha-Synuclein Disease.
Not available
S-EPMC11889527 | biostudies-literature
UnknownThe experience of off periods: Qualitative analysis of interviews with persons with Parkinson's and carepartners.
Not available
S-EPMC8288715 | biostudies-literature
TranscriptomicsIntra- and inter-individual genetic differences in gene expression
Each expressed gene was tested for differential expression in three mouse tissues (Brain, Kidney, Liver) in a direct comparison of SJL/J vs. C57BL/6J Keywords: direct comparison, multiple tissues Whole RNA from a pool of 5 SJL/J mice was compared to a pool of RNA from C57BL/6J mice with three techni...
ORGANISM(S): Mus musculus 
2010-05-16 | E-GEOD-8358 | biostudies-arrayexpress
UnknownAutomated Quality Control for Sensor Based Symptom Measurement Performed Outside the Lab.
Not available
S-EPMC5948536 | biostudies-literature
UnknownHypoxic conditioning in Parkinson's disease: randomized controlled multiple N-of-1 trials.
Not available
S-EPMC12475152 | biostudies-literature
UnknownpH modulates the activity and synergism of the airway surface liquid antimicrobials β-defensin-3 and LL-37.
Not available
S-EPMC4284593 | biostudies-literature
MetabolomicsHuman Gut Microbiota from Autism Spectrum Disorder Promote Behavioral Symptoms in Mice
Autism spectrum disorder (ASD) manifests as alterations in complex human behaviors including social communication and stereotypies. In addition to genetic risks, the gut microbiome differs between typically-developing (TD) and ASD individuals, though it remains unclear whether the microbiome contrib...
2019-07-04 | MTBLS726 | MetaboLights
UnknownMultiple N-of-1 trials to investigate hypoxia therapy in Parkinson's disease: study rationale and protocol.
Not available
S-EPMC9281145 | biostudies-literature
UnknownFamilial knockin mutation of LRRK2 causes lysosomal dysfunction and accumulation of endogenous insoluble α-synuclein in neurons.
Not available
S-EPMC5803451 | biostudies-literature
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