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Transcription is a major obstacle for replication fork progression and a cause of genome instability. Such instability increases in mutants with a suboptimal assembly of the nascent messenger ribonucleo-protein particle (mRNP), as THO/TREX and the NPC-associated THSC/TREX-2 complex. We used microarr...
ORGANISM(S): Saccharomyces cerevisiae 
Transcription is a major obstacle for replication fork progression and a cause of genome instability. Such instability increases in mutants with a suboptimal assembly of the nascent messenger ribonucleo-protein particle (mRNP), as THO/TREX and the NPC-associated THSC/TREX-2 complex. Here we show tha...
ORGANISM(S): Saccharomyces cerevisiae 
Seedlings of Arabidopsis thaliana Col-0 were treated with a cutin oligomeric mixture (COM) for 30 min. To evaluate if such treatment was able to induce transcriptional reprogramming related with plant immunity. The transcriptomic profiles were compared with those of the Mock treatment. The list of d...
ORGANISM(S): Arabidopsis thaliana 
TMT sixplex Isobaric Mass Tagging (TAM) analysis was carried out in the CBMSO Protein Chemistry Facility (ProteoRed, PRB3-ISCIII and UAM University, Spain. Chronic skeletal muscle mitochondria dyshomeostasis drives tubular aggregate formation.
ORGANISM(S): Mus musculus (Mouse) 
2022-08-12 | PXD026771 | Pride
These data show the proteomic profile of the seminal plasma of young Nelore bulls with an average age of 21.7 months and the genetic ontology of the identified proteins.
ORGANISM(S): Cellular Organisms 
Chronic skeletal muscle mitochondria dyshomeostasis drives tubular aggregate formation. TMT sixplex Isobaric Mass Tagging analysis was carried out in the CBMSO Protein Chemistry Facility (ProteoRed, PRB3-ISCIII and UAM University, Spain.
ORGANISM(S): Mus musculus (Mouse) 
2022-08-12 | PXD026722 | Pride
Whether muscle mitochondrial dysfunction is the cause or consequence of metabolic disorders remains controversial. Herein, we demonstrate that inhibition of mitochondrial ATP synthase in muscle in vivo alters whole-body lipid homeostasis. Mice with restrained activity of the enzyme presented intrafi...
ORGANISM(S): Mus musculus (Mouse) 
2020-07-06 | PXD017678 | Pride
Whether muscle mitochondrial dysfunction is the cause or consequence of metabolic disorders remains controversial. Herein, we demonstrate that inhibition of mitochondrial ATP synthase in muscle in vivo alters whole-body lipid homeostasis. Mice with restrained activity of the enzyme presented intrafi...
ORGANISM(S): Mus musculus (Mouse) 
2020-07-06 | PXD017683 | Pride
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