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ProteomicsVAV3 in ERBB4-mediated cancer cell migration
ERBB4 is a member of the epidermal growth factor receptor (EGFR)/ERBB subfamily of receptor tyrosine kinases that regulates cellular processes including proliferation, migration and survival. ERBB4 signaling is involved in embryogenesis and homeostasis of adult tissues, but also in human pathologies...
ORGANISM(S): Homo sapiens (Human) 
2020-06-24 | PXD019430 | Pride
Vav3 Targeted analysis Prm Erbb4 
TranscriptomicsThe Rho Exchange Factors Vav2 and Vav3 Control a Lung MetastasisM-bM-^@M-^SSpecific Transcriptional Program in Breast Cancer Cells
The guanosine triphosphatases of the Rho and Rac subfamilies regulate protumorigenic pathways and are activated by guanine nucleotide exchange factors (Rho GEFs), which could be potential targets for anticancer therapies. We report that two Rho GEFs, Vav2 and Vav3, play synergistic roles in breast c...
ORGANISM(S): Mus musculus 
2012-10-03 | E-GEOD-33348 | biostudies-arrayexpress
GenomicsVAV3 antagonizes Skeletal Muscle Disulfidptosis to maintain Glucose Homeostasis in Type 2 Diabetes
VAV3 antagonizes Skeletal Muscle Disulfidptosis to maintain Glucose Homeostasis in Type 2 Diabetes
PRJNA1164252 | ENA
UnknownVAV3 mediates resistance to breast cancer endocrine therapy.
Not available
S-EPMC4076632 | biostudies-literature
UnknownVav3-deficient mice exhibit a transient delay in cerebellar development.
Not available
S-EPMC2836963 | biostudies-literature
GenomicsNuclear Vav3 is required for canonical PRC1 activity in B-cell lymphoblastic leukemogenesis (RNA-Seq)
Nuclear Vav3 is required for canonical PRC1 activity in B-cell lymphoblastic leukemogenesis (RNA-Seq)
PRJNA804541 | ENA
UnknownVAV3 Gene Polymorphism Is Associated with Paget's Disease of Bone.
Not available
S-EPMC4892207 | biostudies-literature
TranscriptomicsVAV3 antagonizes Skeletal Muscle Disulfidptosis to maintain Glucose Homeostasis in Type 2 Diabetes
The emergence and progression of type 2 diabetes mellitus (T2DM) are intricately linked to skeletal muscle loss, yet the exact mechanisms remain elusive. Here, our whole-exome and transcriptomic results indicate that VAV3 is mutated and down-regulated in T2DM patients associated with increased sulfu...
ORGANISM(S): Homo sapiens 
2025-09-10 | GSE277813 | GEO
GenomicsNuclear Vav3 is required for canonical PRC1 activity in B-cell lymphoblastic leukemogenesis (CUT&RUN)
Nuclear Vav3 is required for canonical PRC1 activity in B-cell lymphoblastic leukemogenesis (CUT&RUN)
PRJNA804543 | ENA
UnknownTargeted overexpression of vav3 oncogene in prostatic epithelium induces nonbacterial prostatitis and prostate cancer.
Not available
S-EPMC2730641 | biostudies-literature
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