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ProteomicsExtracellular vesicle-derived miRNAs improve stem cell-based therapeutic approaches in muscle wasting conditions.
Skeletal muscle holds an intrinsic capability of growth and regeneration both in physiological conditions and in case of injury. Chronic muscle illnesses, generally caused by genetic and acquired factors, lead to deconditioning of the skeletal muscle structure and function, and are associated with a...
ORGANISM(S): Mus musculus (Mouse) 
2022-11-09 | PXD035446 | Pride
Aging Mirna Muscular dystrophy Skeletal muscle Hypertrophy Extracellular vesicle 
UnknownAdoptive Cell Therapy in Pediatric and Young Adult Solid Tumors: Current Status and Future Directions.
Not available
S-EPMC8901720 | biostudies-literature
UnknownGlycogen storage disease type 1a is associated with disturbed vitamin A metabolism and elevated serum retinol levels.
Not available
S-EPMC7001719 | biostudies-literature
UnknownSupra-second interval timing in bipolar disorder: examining the role of disorder sub-type, mood, and medication status.
Not available
S-EPMC10542629 | biostudies-literature
UnknownMutations in LAMB2 causing a severe form of synaptic congenital myasthenic syndrome.
Not available
S-EPMC2643050 | biostudies-literature
MetabolomicsUntargeted metabolomics reveals species-specific metabolite production and shared nutrient consumption by Pseudomonas aeruginosa and Staphylococcus aureus

While bacterial metabolism is known to impact antibiotic efficacy and virulence, the metabolic capacities of individual microbes in cystic fibrosis lung infections are difficult to disentangle from sputum samples. Here, we show that untargeted metabolomic profiling of supernatants of multiple str...

2021-05-26 | MTBLS2105 | MetaboLights
ProteomicsRPE-1 and RPE-1 trisomic AHA p-c and steady state data
We developed a pulse-chase method in where fully SILAC (heavy, medium-heavy and Light) labelled cells were pulsed with Azidohomoalanine (AHA) and then chased for different length of times. In addition, steady state protein levels comparing the RPE- and RPE-1 trisomic cells by standard SILAC labeling...
ORGANISM(S): Homo sapiens (Human) 
2016-10-06 | PXD004915 | Pride
Human Aneuploidy Pre-1 
ProteomicsNIH 3T3 AHA p-c, SILAC p-c, inhibitor, and enrichment control experiments
We developed a pulse-chase method in where fully SILAC (heavy, medium-heavy and Light) labelled cells were pulsed with Azidohomoalanine (AHA) and then chased for different length of times. These experiments were performed with 0, 1, 2, 4, 8, 16 and 13 h of chase. Data from 3 biological replicates ar...
ORGANISM(S): Mus musculus (Mouse) 
2016-10-06 | PXD004929 | Pride
Azidohomoalanine Mouse Silac Degradation Aneuploidy 
ProteomicsA human iPSC-derived sensory neuron platform for high-throughput discovery of neuroprotectants against chemotherapy-induced peripheral neuropathy
Chemotherapy-induced peripheral neuropathy (CIPN) is a major dose-limiting side effect of cancer treatment, yet the lack of predictive human models has hindered therapeutic progress. We have established a scalable model of paclitaxel-induced axon degeneration and neurotoxicity in iPSC-derived sensor...
ORGANISM(S): Homo sapiens (Human) 
2026-04-03 | PXD074954 | Pride
Axon degeneration Ipsc-derived sensory neurons Ste20 kinases Neuroprotective small molecules High-throughput screening Chemotherapy-induced peripheral neuropathy 
UnknownPhage defence by deaminase-mediated depletion of deoxynucleotides in bacteria.
Not available
S-EPMC9830645 | biostudies-literature
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