plasmodium falciparum transmission

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Plasmodium falciparum (66)
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Disease

Plasmodium falciparum malaria (14)
Malaria (9)
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Technology Type

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UNIPROT ID/AC

5833.MAL13P1.267 (2)
5833.MAL13P1.269 (2)
5833.MAL13P1.325 (2)
5833.MAL13P1.334 (2)
5833.MAL7P1.78 (2)
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5833.PF11_0033 (2)
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5833.PF14_0756 (2)
5833.PF14_0759 (2)
5833.PFA0105w (2)
5833.PFA0120c (2)
5833.PFA0705c (2)
5833.PFB0050c (2)
5833.PFB0280w (2)
5833.PFB0925w (2)
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CHEBI ID

CHEBI:71731 (2)
CHEBI:85750 (2)
CHEBI:64498 (2)
CHEBI:85876 (2)
CHEBI:84659 (2)
CHEBI:85743 (2)
CHEBI:84661 (2)
CHEBI:85736 (2)
CHEBI:90302 (2)
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CHEBI:134271 (2)
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CHEBI:134451 (2)
CHEBI:141588 (2)
CHEBI:64516 (2)
CHEBI:134239 (2)
CHEBI:64433 (2)
CHEBI:64520 (2)
CHEBI:71729 (2)
CHEBI:64523 (2)
CHEBI:64424 (2)
CHEBI:134561 (1)
CHEBI:74819 (1)
CHEBI:157889 (1)

Metabolite Name

TAG 56:6 (2)
TAG 54:6 (2)
TAG 54:3 (2)
TAG 54:2 (2)
TAG 52:3 (2)
TAG 52:2 (2)
TAG 52:1 (2)
Myo-Inositol 2-phosphate (1)
(S)-(+)-2-Amino-2-methyl-3-hydroxypropanoic acid (1)
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D-Gentiobiose (1)
(9xi,11beta,14xi)-11,21-Dihydroxypregn-4-ene-3,20-dione (1)
Uridine-diphosphate (1)
Tetramethylethylenediamine (1)
Sterol derivative (1)
Sphingosine (d14:1/22:0) (1)
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Classification

protozoa (73)

Modification

4-tetrahydrothiazine-3-carboxylic acid (1)

Study type

Transcription profiling by array (7)
RNA-seq of coding RNA (4)
Unknown experiment type (2)
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Trajectory (1)
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Release Date

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2007 (10)
2005 (7)
2004 (2)
2002 (2)
1999 (2)
2001 (1)
2000 (1)

Lab affiliation

Department of Microbiology and Molecular Medicine, Faculty of Medicine, University of Geneva, Geneva, CH-1211 Switzerland (2)
Emerging Pathogens Institute, University of Florida (2)
School of Biomedical and Healthcare Sciences; Plymouth University (UK) (2)
-The Walter and Eliza Hall Institute of Medical Research, 1G Royal Parade, Parkville, Victoria 3052, Australia. -Department of Medical Biology, University of Melbourne, Parkville, Victoria 3010, Australia. (1)
Biologie des interactions hôte-parasite Institut Pasteur (1)
Cambridge Institute for Medical Research, University of Cambridge, Cambridge, CB2 0XY, UK (1)
Center for Infection and Immunity of Lille, Inserm U1019-CNRS UMR 8204, Université de Lille, Institut Pasteur de Lille, 1 Rue du Professeur Calmette, F-59000 Lille, France (1)
Center for Molecular Biology of Heidelberg University (ZMBH) (1)
Department of Entomology, Pennsylvania State University, USA (1)
Department of Medical Microbiology, Radboud University Medical Center, The Netherlands (1)
Department of Microbiology, Immunology, and Molecular Genetics (MIMG) Institute for Quantitative and Computational Biosciences (QCBio) University of California, Los Angeles (UCLA) (1)
Department of Molecular Microbiology and Molecular Medicine, Faculty of Medicine, University of Geneva, Switzerland (1)
Dept of Internal Medicine, Morsani College of Medicine, University of South Florida (1)
Division of Infectious Diseases, Boston Children’s Hospital, Boston, MA 02115 Department of Pediatrics, Harvard Medical School, Boston, MA 02115 (1)
EMBL Hamburg Germany (1)
Head Of Proteomics STP, Francis Crick Institute, London (1)
Infectious Diseases, Department of Internal Medicine, Yale School of Medicine, Yale University, New Haven, CT 06520, USA; Howard Hughes Medical Institute, Chevy Chase, MD, 20815, USA (1)
Institute of Structural and Molecular Biology, Birkbeck College, London WC1E 7HX, UK (1)
Italian NationalInstitute of Health (1)
Johns Hopkins Bloomberg School of Public Health (1)
Johns Hopkins School of Medicine (1)
London School of Hygine and Tropical Medicine (1)
Malaria Epigenetics Group Barcelona Institute for Global Health (ISGlobal) Barcelona, Spain (1)
Malaria Immunology Laboratory, The Francis Crick Institute, London, UK (1)
Proteomics and Metabolomics Facility, University of Nebraska - Lincoln (1)
Proteomics platform 3P5-Necker, Université Paris Descartes - Structure Fédérative de Recherche Necker, INSERM US24/CNRS UMS3633, Paris 75014, France. (1)
Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Nijmegen, The Netherlands. (1)
School of Biomedical and Healthcare Sciences Plymouth University Peninsula Schools of Medicine and Dentistry Plymouth University (UK) (1)
The Francis Crick Institute 1 Midland Road London NW1 1AT UK (1)
The Walter and Eliza Hall Institute of Medical Research, 1G Royal Parade, Parkville, Victoria 3052, Australia (1)

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Plasmodium falciparum adapts its investment into replication versus transmission according to the host environment

The malaria parasite life cycle includes asexual replication in human blood, with a proportion of parasites differentiating to gametocytes required for transmission to mosquitoes. Commitment to differentiate into gametocytes, which is marked by activation of the parasite transcription factor ...

2025-07-09 | MTBLS5130 | MetaboLights

Analysis of sex-specific lipid metabolism of Plasmodium falciparum points to the importance of sphingomyelin for gametocytogenesis

Male and female Plasmodium falciparum gametocytes are the parasite lifecycle stage responsible for transmission of malaria from the human host to the mosquito vector. Not only are gametocytes able to survive in radically different host environments, but they are also precursors for male and female g...

2022-04-05 | MTBLS4137 | MetaboLights

Metabolic balancing by miR-276 shapes the mosquito reproductive cycle and Plasmodium falciparum development

The blood-feeding behavior of Anopheles females delivers essential nutrients for egg development and drives parasite transmission between humans. Plasmodium growth is adapted to the vector reproductive cycle, but how changes in the reproductive cycle impact parasite development remains unclear. Here...

2021-11-03 | MTBLS1282 | MetaboLights

Identification of a subtelomeric gene family expressed during the asexual-sexual stage transition in Plasmodium falciparum

For malaria transmission, the parasite must undergo sexual differentiation into mature gametocytes. However, the molecular basis for this critical transition in the parasites life cycle is unknown. Six previously uncharacterized genes, Pfg14.744, Pfg14.745, Pfg14.748, Pfg14.763, Pfg14.752 and Pfg6.6...

ORGANISM(S): Plasmodium falciparum

2014-10-16 | E-GEOD-62364 | biostudies-arrayexpress

Proteomic analyses identified distinct types of vesicles essential for egress of Plasmodium falciparum gametocytes from red blood cells

Transmission of malaria parasites to the mosquito is mediated by sexual precursor cells, the gametocytes. Upon entering the mosquito midgut, the gametocytes egress from the enveloping erythrocyte while passing through gametogenesis. Egress follows an inside-out mode during which the membrane of the ...

ORGANISM(S): Plasmodium Falciparum

2024-01-25 | PXD039679 | JPOST Repository

Plasmodium falciparum W2/Dd2 Day 8 gametocytes

The sexual stages are vital phases in malaria parasite transmission and are the targets of various interventions such as transmission blocking vaccines. The molecular mechanisms underlying sexual development, however, remain poorly understood. We report mappping of a determinant previously linked to...

ORGANISM(S): Plasmodium falciparum

2010-06-10 | E-GEOD-2265 | biostudies-arrayexpress

Histone variant, H2B.Z demarcates the AT-rich promoter regions of the Plasmodium falciparum genome

Histone variants are key components of the epigenetic code and evolved to perform specific functions in transcriptional regulation, DNA repair, chromosome segregation and other fundamental processes. H2B.Z is a rare, apicomplexan-specific variant of histone H2B. Here we show that in Plasmodium falci...

ORGANISM(S): Plasmodium falciparum 3D7

2013-03-05 | E-GEOD-39702 | biostudies-arrayexpress

Expression data from Plasmodium falciparum schizonts

During intra-erythrocytic development, late asexually replicating Plasmodium falciparum parasites sequester from peripheral circulation. This facilitates chronic infection and is linked to severe disease and organ-specific pathology including cerebral and placental malaria. Immature gametocytes M-bM...

ORGANISM(S): Plasmodium falciparum

2015-01-13 | E-GEOD-64887 | biostudies-arrayexpress

Translational regulation of Anopheles gambiae mRNAs in the midgut during Plasmodium falciparum infection

Malaria is caused by Plasmodium parasites, which are transmitted via the bites of infected Anopheline mosquitoes. Midgut invasion is a major bottleneck for Plasmodium development inside the mosquito vectors as a rapidly responding immune system recognizes ookinetes and recruits killing factors from ...

ORGANISM(S): Anopheles gambiae

2012-09-21 | E-GEOD-38707 | biostudies-arrayexpress

P.Falciparum gametocyte egress secretome

Proteins released in the cultured medium of eritrocytes infected by Plasmodium Falciparum at the moment of gametocyte egress

ORGANISM(S): Homo Sapiens (ncbitaxon:9606) Plasmodium Falciparum 3d7 (ncbitaxon:36329)

2020-12-24 | MSV000086635 | MassIVE

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