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ProteomicsIdentification of phosphorylation sites on NAP1/AZI2
We observed smearing for the TBK1-adaptor protein NAP1/AZI2 during mitophagy. To verify whether this smearing was due to phosphorylation by TBK1, we performed an in vitro kinase assay and complemented this with the immunoprecipitation of NAP1 from HAP1, whose genetic background (FIP200 knockout) res...
ORGANISM(S): Homo sapiens (Human) 
2024-04-12 | PXD049184 | Pride
Azi2 Nap1 Mitophagy Tbk1 
UnknownTBK1 adaptor AZI2/NAP1 regulates NDP52-driven mitochondrial autophagy.
Not available
S-EPMC11490886 | biostudies-literature
UnknownTBK1-associated adapters TANK and AZI2 protect mice against TNF-induced cell death and severe autoinflammatory diseases.
Not available
S-EPMC11576971 | biostudies-literature
UnknownAZI2 mediates TBK1 activation at unresolved selective autophagy cargo receptor complexes with implications for CD8 T-cell infiltration in breast cancer.
Not available
S-EPMC10936636 | biostudies-literature
UnknownAZI23'UTR Is a New SLC6A3 Downregulator Associated with an Epistatic Protection Against Substance Use Disorders.
Not available
S-EPMC5886844 | biostudies-literature
Genomics
RNA sequencing from WT or Tank/Azi2 DKO macrophages
PRJNA1095284 | ENA
UnknownFIP200 Suppresses Immune Checkpoint Therapy Responses in Breast Cancers by Limiting AZI2/TBK1/IRF Signaling Independent of Its Canonical Autophagy Function.
Not available
S-EPMC7483892 | biostudies-literature
TranscriptomicsCapture Hi-C reveals novel candidate genes and complex long-range interactions with related autoimmune risk loci
Genome-wide association studies have been tremendously successful in identifying genetic variants associated with complex diseases. The majority of association signals are intergenic and evidence is accumulating that a high proportion of signals lie in enhancer regions.We use Capture Hi-C to investi...
ORGANISM(S): Homo sapiens 
2015-11-24 | E-GEOD-69600 | biostudies-arrayexpress
OtherCapture Hi-C reveals novel candidate genes and complex long-range interactions with related autoimmune risk loci
Genome-wide association studies have been tremendously successful in identifying genetic variants associated with complex diseases. The majority of association signals are intergenic and evidence is accumulating that a high proportion of signals lie in enhancer regions.We use Capture Hi-C to investi...
ORGANISM(S): Homo sapiens 
2015-11-24 | GSE69600 | GEO
UnknownULK1/2 Restricts the Formation of Inducible SINT-Speckles, Membraneless Organelles Controlling the Threshold of TBK1 Activation.
Not available
S-EPMC6710720 | biostudies-literature
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