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UnknownSerum Pepsinogen as a Biomarker for Gastric Cancer in the United States: A Nested Case-Control Study Using the PLCO Cancer Screening Trial Data.
Not available
S-EPMC9268394 | biostudies-literature
TranscriptomicsTranscriptional Regulation of HL60 Neutrophil Differentiation: Promyelocyte trajectory vs. Neutrophil trajectory
Using the HL60 multipotent promyelocytic leukemia cell line, we performed experiments that lead to two different cell fate attractors by treatments of varying dosage and duration of the differentiation agent all-trans-retinoic acid (ATRA). The dosage and duration combinations corresponding to the tw...
ORGANISM(S): Homo sapiens 
2008-11-19 | E-GEOD-13673 | biostudies-arrayexpress
UnknownMarkov-Type State Models to Describe Non-Markovian Dynamics.
Not available
S-EPMC11912194 | biostudies-literature
ProteomicsMyofibroblast-specific Smad7 protects the pressure-overloaded heart by restraining collagen deposition and denaturation, and by inhibiting paracrine macrophage activation
Rationale: Expansion and activation of cardiac fibroblasts contributes to adverse remodeling, fibrosis and dysfunction in the pressure-overloaded heart. Although early fibroblast TGF-β/Smad3 activation protects the pressure overloaded heart by preserving the matrix, sustained TGF-β activation may be...
ORGANISM(S): Mus musculus (Mouse) 
2025-07-28 | PXD043577 | Pride
Heart Tgf-beta Fibrosis Smad7 
UnknownExtracting Pulmonary Nodules and Nodule Characteristics from Radiology Reports of Lung Cancer Screening Patients Using Transformer Models.
Not available
S-EPMC11310180 | biostudies-literature
ProteomicsMyofibroblast-specific Smad7 protects the pressure-overloaded heart by restraining collagen deposition and denaturation, and by inhibiting paracrine macrophage activation
Rationale: Expansion and activation of cardiac fibroblasts contributes to adverse remodeling, fibrosis and dysfunction in the pressure-overloaded heart. Although early fibroblast TGF-β/Smad3 activation protects the pressure overloaded heart by preserving the matrix, sustained TGF-β activation may be...
ORGANISM(S): Mus musculus (Mouse) 
2025-07-28 | PXD049057 | Pride
Heart Proteomics Macrophage Smad7 Pressure 
UnknownCost-Efficient Domain-Adaptive Pretraining of Language Models for Optoelectronics Applications.
Not available
S-EPMC11898057 | biostudies-literature
ProteomicsαKG-mediated carnitine synthesis promotes homologous recombination via histone acetylation
Homologous recombination (HR) deficiency enhances sensitivity to DNA damaging agents commonly used to treat cancer. In HR-proficient cancers, metabolic mechanisms driving response or resistance to DNA damaging agents remain unclear. We identified that depletion of KG sensitizes HR-proficient cells t...
ORGANISM(S): Homo sapiens (Human) 
2026-03-30 | PXD060690 | Pride
Ovarian cancer Epigenetics Dna damaging agents Isocitrate dehydrogenase Glutamine Acetylcarnitine Tmlhe Cyclin e 
UnknownConstruction and application of a protein interaction map for white spot syndrome virus (WSSV).
Not available
S-EPMC3879619 | biostudies-literature
ProteomicsUnbalanced chromatin binding of PRC1 complexes drives neurodevelopmental disorders
Heterozygous de novo missense mutations in genes encoding Polycomb-group (PcG) proteins can cause diverse neurodevelopmental disorders (NDDs), but the underlying mechanisms are not yet understood. Here, we identified novel mutations in the two E3-ligases of the Polycomb Repressive complex 1 (PRC1), ...
ORGANISM(S): Mus musculus (Mouse) 
2026-04-06 | PXD059191 | Pride
Neurodevelopment Rnf2 Prc1 Polycomb Mutations 
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