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TranscriptomicsModulation of miR-29 Expression by Alpha-fetoprotein is linked to the Hepatocellular Carcinoma Epigenome
Globally, hepatocellular carcinoma (HCC) accounts for 70-85% of primary liver cancers and ranks second in the leading cause of male cancer death. Serum alpha-fetoprotein (AFP), normally highly expressed in the liver only during fetal development, is reactivated in 60% of HCC tumors and associated wi...
ORGANISM(S): Homo sapiens 
2014-09-23 | E-GEOD-52018 | biostudies-arrayexpress
ProteomicsIdentification of miR-16/29 targets in adipocytes by LC-MSMS
Cancer cachexia is a multifactorial metabolic syndrome defined by the rapid loss of skeletal muscle mass and the loss of fat mass. Up 80% of cancer patients at the late stage with cachexia suffer from progressive atrophy of adipose tissue. Unlike studies on skeletal muscle wasting, there is limited ...
ORGANISM(S): Mus musculus (Mouse) 
2024-06-13 | PXD037998 | Pride
Lc-msms Mouse Adipocytes Mirnas 
ProteomicsIdentification of miR-16/29 targets in adipocytes by LC-MSMS
Cancer cachexia is a multifactorial metabolic syndrome defined by the rapid loss of skeletal muscle mass and the loss of fat mass. Up 80% of cancer patients at the late stage with cachexia suffer from progressive atrophy of adipose tissue. Unlike studies on skeletal muscle wasting, there is limited ...
ORGANISM(S): Mus musculus (Mouse) 
2024-06-13 | PXD037862 | Pride
Lc-msms Mouse Adipocytes Mirnas 
GenomicsExpression data from mouse skin treated with a miR-29 mimic or a miR-29 inhibitor
Expression data from mouse skin treated with a miR-29 mimic or a miR-29 inhibitor
PRJNA438999 | ENA
TranscriptomicsGlobal profiling of gene expression in mouse astrocyte in response to the potential longevity determinant miR-29
MicroRNAs (miRNAs) are an evolutionarily conserved large class of small non-coding RNAs that mediate post-transcriptional silencing of genes and influence a broad spectrum of biological processes ranging from embryonic development to organismal death. Our previous study identified the miR-29 family,...
ORGANISM(S): Mus musculus 
2011-02-08 | E-GEOD-27035 | biostudies-arrayexpress
GenomicsMAPK-induced miR-29 targets MAFG and suppresses melanoma progression
MAPK-induced miR-29 targets MAFG and suppresses melanoma progression
PRJNA625032 | ENA
GenomicsmiR-29 is an important driver of aging-related phenotypes
miR-29 is an important driver of aging-related phenotypes
PRJNA893541 | ENA
ProteomicsMouse Smooth muscle cell secretome with Anti-miR / pre-miR treatment
Overexpression and inhibition of miR-29 (pre-miR and anti-miR to miR-29b) in murine aortic smooth muscle cells, analysis of their secretome (conditioned media after serum starvation), n=3 for all four groups (pre-miR control, pre-miR-29b, anti-miR control, anti-miR-29b).
ORGANISM(S): Mus musculus (Mouse) 
2016-05-06 | PXD003938 | Pride
Mouse Smooth muscle cell Mir-29 
GenomicsmiR-29 controls aging-related phenotypes through regulation of the p53 signaling pathway
miR-29 controls aging-related phenotypes through regulation of the p53 signaling pathway
PRJNA421320 | ENA
TranscriptomicsmiR-29 is an important driver of aging-related phenotypes
Aging is a consequence of complex molecular changes, but the roles for individual microRNAs (miRNAs) in aging remain unclear. One of the few miRNAs that is upregulated during both normal and premature aging is miR-29. We confirmed this finding in our study in both mouse and monkey models. Follow-up ...
ORGANISM(S): Mus musculus 
2025-10-22 | GSE216398 | GEO
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