Sort   by:  
 Page size 
TranscriptomicsTranscription profile of adipose derived mesenchymal stem cells separated by cell surface protein N-cadherin.
Mesenchymal stem cells (MSCs) are one of most promising stem cell sources for regenerative medicine. MSCs have a differention ability into several tissues. However, their differentiation efficiency is considered quite limited. Especially, the differentiation efficiency into cardiomyocytes is very lo...
ORGANISM(S): Homo sapiens 
2013-10-29 | E-GEOD-35232 | biostudies-arrayexpress
OtherThe N-cadherin interactome in primary cardiomyocytes
Low levels of Cdh2-BioID2 were expressed in mouse primary neonatal cardiomyocytes using an adenoviral expression system. Infected cells were treated with biotin for 24 hours before lysis. Biotinylated proteins were precipitated from lysates using streptavidin beads. Protein samples were in-gel tryps...
ORGANISM(S): Mus Musculus 
2019-01-23 | MSV000083356 | MassIVE
TranscriptomicsTranscription profiling of mouse hematopoetic progenitor cells with Flk12+ Ncad-, Flk12- Ncad+, Flk12+ Ncad+ or Flk12- Ncad- phenotypes
N-cadherin expression level distinguishes reserved versus primed states of hematopoietic stem cells
ORGANISM(S): Mus musculus 
2008-01-04 | E-TABM-404 | biostudies-arrayexpress
ProteomicsThe N-cadherin interactome in primary cardiomyocytes as defined by quantitative proximity proteomics
Low levels of Cdh2-BioID2 were expressed in mouse primary neonatal cardiomyocytes using an adenoviral expression system. Infected cells were treated with biotin for 24 hours before lysis. Biotinylated proteins were precipitated from lysates using streptavidin beads. Protein samples were in-gel tryps...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
2019-01-24 | MSV000083362 | MassIVE
TranscriptomicsRac1-Induced Connective Tissue Growth Factor regulates Connexin 43 and N-Cadherin Expression in Atrial Fibrillation
Objectives: We studied the signal transduction of atrial structural remodelling that contributes to the pathogenesis of atrial fibrillation (AF). Backround: Fibrosis is a hallmark of arrhythmogenic structural remodelling but the underlying molecular mechanisms are incompletely understood. Methods: W...
ORGANISM(S): Homo sapiens 
2010-02-21 | E-GEOD-14975 | biostudies-arrayexpress
ProteomicsMapping of phosphorylation target sites of obscurin-kin 1 within the cytoplasmic domain of N-cadherin by LC-MS/MS
To test our hypothesis that obscurin-kin1 modulates, at least in part, the mechanochemical coupling of cardiomyocytes via phosphorylation of N-cadherin, which has been shown to play a pivotal role in these processes, we set forth to identify the potential phosphorylation target sites of obscurin-kin...
ORGANISM(S): Mus musculus (Mouse) 
2024-05-23 | PXD031003 | Pride
Cytoplasmic domain of n-cadherin Obscurin kinase 1 Ser phosphorylation Lc-ms/ms 
GenomicsN-cadherin antagonism is bronchoprotective in severe asthma models
N-cadherin antagonism is bronchoprotective in severe asthma models
PRJNA1178626 | ENA
ProteomicsSwarms of specialized fibroblasts drive scarring through N-cadherin
Injury in mammals induces a connective tissue response to either regenerate as before, or to scar. The fibroblastic actions and mechanisms that underlie these connective tissue responses remain obscure, as direct observation in animals is too difficult and current assays do not faithfully reproduce ...
ORGANISM(S): Mus musculus (Mouse) 
2020-10-26 | PXD016068 | Pride
Live imaging Collective migration Fibroblastic swarm Cell lineage Contraction Scad Scarring Scar progenitors N-cadherin 
ProteomicsPomGnt1 deficiency affects N-cadherin mediated cell-cell adhesion
N-glycoproteomic and O-man glycoproteomic analysis of N-cadherin derived from wild-type(WT) and POMGnT1-deficient HEK293T cells. Proteolytic digestion was performed with trypsin or proteinase K. Glycopeptide enrichment was achieved using cotton-HILIC SPE. Glycopeptides were fragmented by HCD.step an...
ORGANISM(S): Human 
2020-04-07 | MSV000085243 | MassIVE
GenomicsN-Cadherin promotes cardiac regeneration by potentiating pro-mitotic β-Catenin signaling in cardiomyocytes
N-Cadherin promotes cardiac regeneration by potentiating pro-mitotic β-Catenin signaling in cardiomyocytes
PRJNA1068015 | ENA
Sort   by:  
 Page size