Staab-Weijnitz Claudia C

Show results for

Proteomics (3)

Organisms

Alphavirus (3)
Anemia (3)
Archaea (3)
Bacteria (3)
Betaherpesvirinae (3)
Borrelia (3)
Chlorella vulgaris (3)
Clostridium perfringens (3)
Dengue virus (3)
Enterovirus A71 (3)
Eukaryota (3)
Hepatitis B virus (3)
Human alphaherpesvirus 1 (3)
Human betaherpesvirus 5 (3)
Influenza A virus (3)
Influenza C virus (3)
Japanese encephalitis virus group (3)
Kamptonema sp. (3)
Listeria monocytogenes (3)
Mammarenavirus (3)
Miscanthus x giganteus (3)
Mycobacterium tuberculosis (3)
Orthoflavivirus (3)
Panicum virgatum (3)
Pseudomonas aeruginosa (3)
Rattus (3)
Severe acute respiratory syndrome coronavirus 2 (3)
Staphylococcus aureus (3)
Streptococcus pneumoniae (3)
Viruses (3)

Repository

pride (3)

Tissue

Lung (2)
Epithelial cell (1)

Disease

Idiopathic pulmonary fibrosis (1)

Technology Type

Mass Spectrometry (3)
Shotgun proteomics (2)
Bottom-up proteomics (1)

Publication Date

2026 (1)
2025 (1)
2022 (1)

Lab affiliation

Institute of Lung Health and Immunity (LHI), Helmholtz Munich, Comprehensive Pneumology Center (CPC-M), Germany, Member of the German Center for Lung Research (DZL); Department of Pediatrics, University of Colorado Anschutz Medical Campus, Aurora, CO, USA (1)
Institute of Lung Health and Immunity and Comprehensive Pneumology Center with the CPC-M BioArchive, Helmholtz Zentrum München, Member of the German Center of Lung Research (DZL) - München (Germany) (1)
Institute of Lung Health and Immunity, and Comprehensive Pneumology Center with the CPC-M bioArchive, Ludwig-Maximilians-Universität München and Helmholtz Zentrum München, Munich, Germany, Member of the German Center for Lung Research (DZL); 8Department of Pediatrics, and Division of Pulmonary Sciences and Critical Care Medicine, School of Medicine, University of Colorado Anschutz Medical Campus, Aurora, CO, USA (1)

Previous page
1 / 1
You're on page 1
Next page

Sort by:

Relevance

Page size

Quantitative proteomics of airway epithelial cells from COPD and control pre- and post-influenza virus infection

Chronic obstructive pulmonary disease (COPD) collectively refers to chronic and progressive lung diseases causing not fully reversible limitations in airflow. COPD patients are at high risk for severe respiratory symptoms upon influenza virus infection. Airway epithelial cells provide the first-line...

ORGANISM(S): Homo sapiens (Human)

2022-12-29 | PXD031461 | Pride

Site-specific quantification of collagen post-translational modifications reveals profound disruption of the collagen biosynthetic network upon prolyl-3-hydroxylase 1 deficiency

Type I collagen is the main structural protein in mammals and other vertebrate life forms. During the biosynthesis of collagens, prolyl-3-hydroxylase 1 (P3H1) catalyzes 3-hydroxylation of proline residues and acts as a chaperone. Deficiency of P3H1 causes Osteogenesis Imperfecta (OI), a hereditary d...

ORGANISM(S): Mus musculus (Mouse)

2026-04-02 | PXD053751 | Pride

Glycosylation Collagen Tendon Post-translational modification (ptm) Prolyl-3-hydroxylase Hydroxyproline

COLLAGEN POST-TRANSLATIONAL MODIFICATIONS ARE ALTERED IN IPF INCLUDING WITHIN ECM RECEPTOR BINDING MOTIFS

Idiopathic pulmonary fibrosis (IPF) is a fatal chronic lung disease with a median survival of 3.8 years, a prevalence of 10 to 60 cases per 100,000 in the United States, and rising incidence and mortality rates. Currently approved antifibrotic agents can slow disease progression but are insufficient...