Sort   by:  
 Page size 
UnknownThe bacterial cytoskeleton: more than twisted filaments.
Not available
S-EPMC3597445 | biostudies-literature
UnknownAnti-tuberculosis drug resistance in Slovakia, 2018-2019: The first whole-genome epidemiological study.
Not available
S-EPMC8717600 | biostudies-literature
UnknownOptimizing the Use of Gene Expression Profiling in Early-Stage Breast Cancer.
Not available
S-EPMC5455310 | biostudies-literature
UnknownKinetics of Genetic Variation of the Mycoplasma genitalium MG192 Gene in Experimentally Infected Chimpanzees.
Not available
S-EPMC4771357 | biostudies-literature
UnknownMycoplasma genitalium: an efficient strategy to generate genetic variation from a minimal genome.
Not available
S-EPMC2169797 | biostudies-literature
ProteomicsOptimizing PSC culture for generating neural organoids
Cerebral organoids generated according to unguided protocols produce neural tissue with exceptional cell diversity and fidelity to in vivo. However, with only minimal extrinsic intervention, the importance of high quality starting material becomes paramount. Better understanding of what constitutes ...
ORGANISM(S): Homo sapiens (Human) 
2025-10-27 | PXD061650 | Pride
Lc-msms Proteomics Phospho Pluripotent stem cells 
ProteomicsProteomic analysis of human PARK2 knockout iPSC-derived neurons
Mitochondrial dysfunction plays a major role in the pathogenesis of sporadic Parkinson’s disease (PD) and familial PD caused by mutations in the PARK2 gene. The protein, parkin, is vital for mitochondrial function, but the lack of key PD phenotypes in PARK2 knockout (KO) rodent models has hindered i...
ORGANISM(S): Homo sapiens (Human) 
2019-09-03 | PXD007871 | Pride
Lc-msms Qexactive-hf Parkinson's disease Human ips cells 
UnknownGenetic variation in the complete MgPa operon and its repetitive chromosomal elements in clinical strains of Mycoplasma genitalium.
Not available
S-EPMC3004944 | biostudies-literature
ProteomicsProteomic characterisation of lysosomal and mitochondrial perturbations in human iPSC-derived neurons with PARK2 mutation
Mitochondrial dysfunction plays a major role in the pathogenesis of sporadic Parkinson’s disease (PD) and familial PD caused by mutations in the PARK2 gene. The protein, parkin, is vital for mitochondrial function, but the lack of key PD phenotypes in PARK2 knockout (KO) rodent models has hindered i...
ORGANISM(S): Homo sapiens (Human) 
2019-07-26 | PXD008894 | Pride
Human Lc-msms Ipsc Tmt Park2 Mitochondria 
UnknownShort tandem repeat sequences in the Mycoplasma genitalium genome and their use in a multilocus genotyping system.
Not available
S-EPMC2515158 | biostudies-literature
Sort   by:  
 Page size