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TranscriptomicsEffects of 30 days resveratrol supplementation on adipose tissue morphology and gene expression patterns in obese men
Polyphenolic compounds, such as resveratrol, have recently received widespread interest due to their ability to mimic effects of calorie restriction. The objective of the present study was to gain more insight into the effects of 30 days resveratrol supplementation on adipose tissue morphology and u...
ORGANISM(S): Homo sapiens 
2013-09-25 | E-GEOD-42432 | biostudies-arrayexpress
Proteomicsamyotrophic lateral sclerosis proteomic signature and treatment with mesenchymal stem cell-derived extracellular vesicles
identifying the proteomic signature of ALS and how this is changed after treatment with MSC-EVs
ORGANISM(S): Homo sapiens (Human) 
2026-09-14 | PXD034824 | Pride
Proteomics Mesenchymal stem cells Extracellular vesicles Amyotrophic lateral sclerosis 
UnknownPrimary Results From the Understanding Outcomes With the S-ICD in Primary Prevention Patients With Low Ejection Fraction (UNTOUCHED) Trial.
Not available
S-EPMC7752215 | biostudies-literature
ProteomicsAlternative mRNA splicing controls functions of the human H3K27 demethylase UTX/KDM6A
The UTX/KDM6A gene encodes the UTX histone H3K27 demethylase, which plays an important role in mammalian development and is frequently mutated in cancers and particularly, in urothelial cancers. Using BioID technique, we explored the interactome of different UTX isoforms.
ORGANISM(S): Homo sapiens (Human) 
2024-01-28 | PXD041254 | Pride
Isoforms Kdm6a Bladder cancer 
ProteomicsMenin-MLL inhibitors enhance AP-1 transcription factor JUND activity in MLLr leukemic cells which contributes to tumorigenesis and therapy resistance (sampleIDs - EO241-EO252; MV-411 cell lines)
Background: MLL-rearranged (MLLr) acute myeloid leukemia (AML) and acute lymphoid leukemia (ALL) involve reciprocal oncogenic translocations of the KMT2A/KMT2B (MLL1/MLL2) gene on chromosome 11q23 with various translocation partner genes, which yields to chimeric MLL fusion proteins (MLL-FPs). Menin...
ORGANISM(S): Homo sapiens (Human) 
2026-07-28 | PXD067825 | Pride
Menin  jund  chromatin modifiers  epigenetic  mllr leukemia  vtp-50469 
ProteomicsMenin-MLL inhibitors enhance AP-1 transcription factor JUND activity in MLLr leukemic cells which contributes to tumorigenesis and therapy resistance (sampleIDs - EO265_276; MOLM13 cell lines)
Background: MLL-rearranged (MLLr) acute myeloid leukemia (AML) and acute lymphoid leukemia (ALL) involve reciprocal oncogenic translocations of the KMT2A/KMT2B (MLL1/MLL2) gene on chromosome 11q23 with various translocation partner genes, which yields to chimeric MLL fusion proteins (MLL-FPs). Menin...
ORGANISM(S): Homo sapiens (Human) 
2026-07-28 | PXD068124 | Pride
Menin  jund  chromatin modifiers  epigenetic  mllr leukemia  vtp-50469 
ProteomicsMenin-MLL inhibitors enhance AP-1 transcription factor JUND activity in MLLr leukemic cells which contributes to tumorigenesis and therapy resistance (sampleIDs - EO85_120; HeLa cell lines)
Background: MLL-rearranged (MLLr) acute myeloid leukemia (AML) and acute lymphoid leukemia (ALL) involve reciprocal oncogenic translocations of the KMT2A/KMT2B (MLL1/MLL2) gene on chromosome 11q23 with various translocation partner genes, which yields to chimeric MLL fusion proteins (MLL-FPs). Menin...
ORGANISM(S): Homo sapiens (Human) 
2026-07-28 | PXD068061 | Pride
Menin  jund  chromatin modifiers  epigenetic  mllr leukemia  vtp-50469 
ProteomicsMenin-MLL inhibitors enhance AP-1 transcription factor JUND activity in MLLr leukemic cells which contributes to tumorigenesis and therapy resistance (sampleIDs - EO01_85; HeLa cell lines)
Background: MLL-rearranged (MLLr) acute myeloid leukemia (AML) and acute lymphoid leukemia (ALL) involve reciprocal oncogenic translocations of the KMT2A/KMT2B (MLL1/MLL2) gene on chromosome 11q23 with various translocation partner genes, which yields to chimeric MLL fusion proteins (MLL-FPs). Menin...
ORGANISM(S): Homo sapiens (Human) 
2026-07-28 | PXD068056 | Pride
Menin  jund  chromatin modifiers  epigenetic  mllr leukemia  vtp-50469 
ProteomicsHistone peptide pull-down H3K4me0-3 +/- H3Q5ser
The interactome to H3 peptides in different H3K4 methylation states and its change upon H3Q5 serotonylation was determined by performing peptide pull-down followed by qMS. Streptaviding beads, saturated with biotinylated H3 peptides in all possible H3K4 methylation states and with presence and absen...
ORGANISM(S): Homo sapiens (Human) 
2025-06-10 | PXD062109 | Pride
Hela H3 H3q5 
ProteomicsgreenCUT&RUN: a rapid and efficient genome profiling approach for transcription and chromatin regulatory proteins
Chromatin modifiers, transcription factors and cofactors regulate gene expression through engagement with chromatin and DNA, which can be detected by a variety of genomic techniques. Here, we describe a novel method ‘greenCUT&RUN’ for genome-wide profiling of transcription regulatory factors, which ...
ORGANISM(S): Homo sapiens (Human) 
2021-01-13 | PXD021089 | Pride
Affinity purification Nanobody Nfya Fos Gfp-tagged proteins Hela Enhancer 
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